AHMAS

Thema: ISPOG

• A. Knowledge
• B. Methodical work
• C. Meta compentencies
• D. Demo VSD
  • Introduction
  • 1. Definition & Aims
  • 1.1 Specific aims
  • 1.2 General aims
  • 2. Case history
  • 3. Didactical questions
  • 3.1 Facts & Figures
  • 3.2 Aetiology
  • 3.3 Critical reflection
  • 3.4 Competency profile
  • 4. Facts & Figures
  • 4.1 Formal definition
  • 4.2 Prevalence
  • 5. Aetiology & Pathology
  • 5.1 Risk factors
  • 5.2 Onset characteristics
  • 5.3 Pathology
  • 5.4 Working model
  • 6. Treatment summary
  • 6.1 Orientation
  • 6.2 Diagnostic phase
  • 6.3 Indication phase
  • 6.4 Informed consent
  • 6.5 Therapeutic phase
  • 6.6 Evaluation
  • 7 Competency profile
  • 7.1 Expert
  • 7.1.1 Consultant
  • 7.1.2 Knowledge
  • 7.1.3 Pat.assessment
  • 7.1.4 Effectiveness
  • 7.1.5 Procedures
  • 7.1.6 Consultation others
  • 7.2 Communicator
  • 7.2.1 Rapport
  • 7.2.2 Relevant info
  • 7.2.3 Patients
  • 7.2.4 Understanding
  • 7.2.5 Information
  • 7.3 Collaborator
  • 7.3.1 Participation
  • 7.3.2 Work together
  • 7.4 Manager
  • 7.4.1 Participation
  • 7.4.2 Self management
  • 7.4.3 Allocation
  • 7.4.4 Leadership
  • 7.5 Health advocate
  • 7.5.1 Individual needs
  • 7.5.2 Communities
  • 7.5.3 Populations
  • 7.5.4 Promotion
  • 7.6 Scholar
  • 7.6.1 Ongoing learning
  • 7.6.2 Evaluate
  • 7.6.3 Facilitate
  • 7.6.4 Contribution
  • 7.7 Professional
  • 7.7.1 Commitment
  • 7.7.2 Participation
  • 7.7.3 Sustainability
• E. E-teaching
• F. ISPOG2019

5.1 Risk factors

The following risk factors are described: 

• Genetic disposition; PVD is linked to genetic patterns that have a high level of infection and whiteness of the skin;
• Neural functioning and density; the function of nerve vessels in the vestibulum seems disturbed, possibly as a reaction to the high infection rates. The elevated nerve density (x10!) may lead to hyper sensitivity and a vicious circle of pain-proliferation-pain etc.;
• Hormones; hormonal variables may play a role because oral anti conception coincides with a relative risk of 6.3 on PVD (9.3 if started with OAC before 16 years old);
• Sensitization; sensitization for sperm has been reported;
• Ethnicity; PVD is hardly seen in non-western populations; this however is no guarantee for a racial protection mechanism;
• Age; PVD used to be seen as a young women disease, however recent research has shown that it also occurs in older women.